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Expression of pain-related molecules in the spinal cord. (A) In the SCI model, <t>p-p38</t> and p-ERK1/2 expression colocalized with CD11b at the lesion site peaked at 2 weeks post-injury and increased in the lumbar enlargement during the chronic phase (12 weeks). Scale bars = 50 μm. (B) In the DCM model, expression of p-p38 and p-ERK1/2 colocalized with CD11b progressively increased at the compression site, with little change in the lumbar enlargement. Scale bars = 50 μm.
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Expression of pain-related molecules in the spinal cord. (A) In the SCI model, <t>p-p38</t> and p-ERK1/2 expression colocalized with CD11b at the lesion site peaked at 2 weeks post-injury and increased in the lumbar enlargement during the chronic phase (12 weeks). Scale bars = 50 μm. (B) In the DCM model, expression of p-p38 and p-ERK1/2 colocalized with CD11b progressively increased at the compression site, with little change in the lumbar enlargement. Scale bars = 50 μm.
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Expression of pain-related molecules in the spinal cord. (A) In the SCI model, <t>p-p38</t> and p-ERK1/2 expression colocalized with CD11b at the lesion site peaked at 2 weeks post-injury and increased in the lumbar enlargement during the chronic phase (12 weeks). Scale bars = 50 μm. (B) In the DCM model, expression of p-p38 and p-ERK1/2 colocalized with CD11b progressively increased at the compression site, with little change in the lumbar enlargement. Scale bars = 50 μm.
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https://www.bioz.com/result/anti phospho p38 map kinase/product/Cell Signaling Technology Inc
Average 86 stars, based on 1 article reviews
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Expression of pain-related molecules in the spinal cord. (A) In the SCI model, <t>p-p38</t> and p-ERK1/2 expression colocalized with CD11b at the lesion site peaked at 2 weeks post-injury and increased in the lumbar enlargement during the chronic phase (12 weeks). Scale bars = 50 μm. (B) In the DCM model, expression of p-p38 and p-ERK1/2 colocalized with CD11b progressively increased at the compression site, with little change in the lumbar enlargement. Scale bars = 50 μm.
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https://www.bioz.com/result/p38 mapk activation/product/MedChemExpress
Average 95 stars, based on 1 article reviews
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Expression of pain-related molecules in the spinal cord. (A) In the SCI model, <t>p-p38</t> and p-ERK1/2 expression colocalized with CD11b at the lesion site peaked at 2 weeks post-injury and increased in the lumbar enlargement during the chronic phase (12 weeks). Scale bars = 50 μm. (B) In the DCM model, expression of p-p38 and p-ERK1/2 colocalized with CD11b progressively increased at the compression site, with little change in the lumbar enlargement. Scale bars = 50 μm.
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Average 95 stars, based on 1 article reviews
p38 map kinase inhibitor iii - by Bioz Stars, 2026-06
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Expression of pain-related molecules in the spinal cord. (A) In the SCI model, p-p38 and p-ERK1/2 expression colocalized with CD11b at the lesion site peaked at 2 weeks post-injury and increased in the lumbar enlargement during the chronic phase (12 weeks). Scale bars = 50 μm. (B) In the DCM model, expression of p-p38 and p-ERK1/2 colocalized with CD11b progressively increased at the compression site, with little change in the lumbar enlargement. Scale bars = 50 μm.

Journal: Frontiers in Cellular Neuroscience

Article Title: Differential microglial dynamics and neuroinflammation underlying neuropathic pain in the central nervous system: comparative insights from spinal cord injury and compressive myelopathy models

doi: 10.3389/fncel.2026.1769004

Figure Lengend Snippet: Expression of pain-related molecules in the spinal cord. (A) In the SCI model, p-p38 and p-ERK1/2 expression colocalized with CD11b at the lesion site peaked at 2 weeks post-injury and increased in the lumbar enlargement during the chronic phase (12 weeks). Scale bars = 50 μm. (B) In the DCM model, expression of p-p38 and p-ERK1/2 colocalized with CD11b progressively increased at the compression site, with little change in the lumbar enlargement. Scale bars = 50 μm.

Article Snippet: The primary antibodies (Abs) used were rabbit anti-phospho-p38 MAP kinase (p-p38) polyclonal Ab (1:400, Cell Signaling Technology, Beverly, MA); rabbit anti- p44/42 MAPK (p-ERK1/2) polyclonal Ab (Cell Signaling Technology); and rabbit anti-CD11b Ab (1:50, Abcam, Cambridge, UK).

Techniques: Expressing

Expression of pain-related molecules in the brain. (A) In the SCI model, there was significant expression of p-p38 and p-ERK1/2 colocalized with CD11b in the hippocampus and amygdala during the chronic phase (12 weeks) and in the thalamus at both acute and chronic time points. Scale bars = 50 μm. (B) In the DCM model, expression of pain-related molecules increased in the hippocampus, amygdala, and thalamus under moderate compression (18 weeks). Scale bars = 50 μm.

Journal: Frontiers in Cellular Neuroscience

Article Title: Differential microglial dynamics and neuroinflammation underlying neuropathic pain in the central nervous system: comparative insights from spinal cord injury and compressive myelopathy models

doi: 10.3389/fncel.2026.1769004

Figure Lengend Snippet: Expression of pain-related molecules in the brain. (A) In the SCI model, there was significant expression of p-p38 and p-ERK1/2 colocalized with CD11b in the hippocampus and amygdala during the chronic phase (12 weeks) and in the thalamus at both acute and chronic time points. Scale bars = 50 μm. (B) In the DCM model, expression of pain-related molecules increased in the hippocampus, amygdala, and thalamus under moderate compression (18 weeks). Scale bars = 50 μm.

Article Snippet: The primary antibodies (Abs) used were rabbit anti-phospho-p38 MAP kinase (p-p38) polyclonal Ab (1:400, Cell Signaling Technology, Beverly, MA); rabbit anti- p44/42 MAPK (p-ERK1/2) polyclonal Ab (Cell Signaling Technology); and rabbit anti-CD11b Ab (1:50, Abcam, Cambridge, UK).

Techniques: Expressing